article · International Journal of Biological Macromolecules
The novel point is that these results demonstrated that the targeted Silymarin NPs not only could efficiently alleviate CCl<sub>4</sub>-induced liver fibrosis more than using only free silymarin; by inhibiting the TGF-β/Smad-3 signaling pathway, but also could activate hepatic progenitor stem cells causing liver regeneration.
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DOI: 10.1016/j.ijbiomac.2025.142837
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